Saturday, February 5, 2011
Kill Cancer By New Tech: Radiology application for mobile devices...WOW!: "Provides wireless access to medical images for iPhone, iPad users FDA clears first diagnostic radiology application for mobile devices,..."
Posted by Nasser at 12:38 PM
|Provides wireless access to medical images for iPhone, iPad users|
FDA clears first diagnostic radiology application for mobile devices, A new mobile radiology application cleared today by the U.S. Food and Drug Administration will allow physicians to view medical images on the iPhone and iPad manufactured by Apple Inc.
“This important mobile technology provides physicians with the ability to immediately view images and make diagnoses without having to be back at the workstation or wait for film,” said William Maisel, M.D., M.P.H., chief scientist and deputy director for science in the FDA’s Center for Devices and Radiological Health.
Posted by Nasser at 12:35 PM
Thursday, February 3, 2011
You may do not know that drugs can disrupt the function of receptors on a cell's surface. Breast cancer – the most prevalent cancer affecting women – is being successfully treated using these "receptor blockers." many cancers actually require hormones to grow. Breast cancer is a very good example. Breast cancer arises in the breast, and the breast is an organ that is responsive to the female hormone, estrogen.
Cancers derived in the breast, therefore, especially in older women, tend to require estrogen to grow. If we can starve that cell of estrogen, we can actually make that cancer cell die.
One of the drugs is called tamoxifen, which actually attaches to the estrogen receptor.
The estrogen receptor is a protein that is found in many breast cancer cells that finds estrogen in surrounding blood, takes it into the cell, and signals the cell to grow.
Well, tamoxifen attaches to the estrogen receptor so the estrogen can’t attach to the receptor. And when tamoxifen and the receptor for estrogen go into the nucleus of the cell, it attaches to the DNA and actually signals the cell not only to grow but to die. So that’s a very important molecule.
One of the really important things to know about cancer is that no two cancers are the same. They're all different. There is tremendous variations in the molecules that are involved in making a cell cancerous. By identifying those molecules in the individual case, therapy can be individualized – give people the medicines they need, and not give medicines to people if they are not going to benefit.The estrogen receptor is not the only target in the treatment of breast cancer. Another important target is the human epidermal growth factor receptor (Her-2), which is over-expressed in 25% of breast cancers, leading to cellular growth and proliferation.
If a cell has too much her2, the cell will be dividing too often because the cell will be interpreting many stimuli in the environment as stimuli cause it to divide whereas a normal cell wouldn’t. It makes that cell have too much of a tendency for cell division and to go on to form a lump and to go on and spread into the surrounding tissue like an invasion or to grow on other parts of the body.
We've developed an antibody that attaches to her2. Antibodies are the proteins that your body makes naturally in response to infections. And your body normally makes lots of antibodies, and should be that's how come you fight lots of infections. However, her2 antibodies can be made – we call it Trastuzumab or Herceptin – outside the human body and give it to the patient intravenously if that patient has a cancer, particularly a breast cancer, with too much her2 in it.
Herceptin is one example of a treatment that targets a specific molecule in a particular type of cancer. As more of these precisely targeted therapies are developed, it will become increasingly important to understand which molecules play important roles in a particular individual’s cancer.
Larry Norton, M.D
Posted by Nasser at 10:15 AM
Wednesday, February 2, 2011
Unlike normal cells, cancer cells can grow and age without dying — one of the reasons they’re so dangerous. But researchers at Washington State University have developed a way to help cancer cells age and die, which could lead to treatment that slows or stops tumor growth.
How do cancer cells differ from normal cells in terms of their mortality?
The big difference between cancer cells and normal cells is that cancer cells can divide forever and live forever. We call this immortality. The normal cells will divide for a number of divisions and then stop growing. They get old and either they die or they sit there and do nothing. They are mortal.
Cancer cells have a way to maintain their . Their telomeres don’t get shortened. Each time the normal cells divide they lose some telomere DNA sequences. Eventually when the telomere DNA becomes too short, they stop growing. There are also other factors contributing to the mortality of normal cells.
Is the immortality of cancer cells what makes them so dangerous?
The cancer cells divide uncontrollably. Then you have more and more cancer cells in one location of your body that can invade the surrounding tissues and disrupt the function of the normal tissues. They form the tumor. The cancer cells also can circulate around your body and get into other places and form tumors in the new locations. This is in part due to the immortality of cancer cells. They don’t die. Normal cells grow at one location and at some point they will stop.
Posted by Nasser at 8:03 AM
Tuesday, February 1, 2011
Introducing a shielded door that knocks current technology off its hinges.
- Smoother, safer operation backed by 100% lifetime guarantee
- Maintenance-free operation
- 12-second open/close—more than twice as fast as competing doors
- Smart Door operational technology allows you to set your own user parameters
- Advanced touch-screen operation controls every aspect of door operation from a single interface
- Remote open/close—control door operation from nurses' station or other area
- Thumbprint ID technology option—eliminate unauthorized access
Posted by Nasser at 8:43 AM
External mark setup introduces considerable target uncertainty resulting from internal organ motion and patient
position variability. ACCULOC IGRT eliminates this uncertainty using implanted gold fiducials or anatomic landmarks
with EPID or CR images. The 3D transformation software (ISOLOC™) outputs exact couch translations for highaccuracy, verifiable localization.
In addition, ACCULOC has the versatility
to support image pairs at any angle -
nonorthogonal images can accommodate larger
patients, hip replacements and other challenges.
For more information, please follow this link: www.civco.com
Posted by Nasser at 6:48 AM
Monday, January 31, 2011
A system and method for directly measuring dose deposition during radiation therapy has been developed by the Mayo Foundation for Medical Education and Research (Rochester, MN). The system uses a Compton camera imager to image photons that have been ejected from the subject due to Compton scattering induced by the radiation treatment. The filing also details a method for reconstructing the acquired Compton scatter photon data to produce an image that shows dose deposition in the subject (WO/2010/141583).medicalphysicsweb.org
Posted by Nasser at 12:48 PM
Kill Cancer By New Tech: External Radiation Therapy: "What Is Radiation Therapy? Cancer is a disease that causes cells to grow abnormally and out of control. In radiation therapy, high-e..."
Posted by Nasser at 7:02 AM
What Is Radiation Therapy?
While it's killing the cancer, radiation therapy also can damage normal cells. The good news is that normal cells are more likely to recover from the effects of radiation. Doctors take precautions to protect a person's healthy cells when they're giving radiation treatments.
Sunday, January 30, 2011
|Different types of gold markers|
Gold Soft Tissue Markers are pure gold cylinders specially knurled to inhibit migration. Standard Gold Soft Tissue Markers are 3mm long and 0.8, 0.9, 1.2 or 1.6mm in diameter. They are available as loose markers or in sterilized preloaded needles for ease of use.
The use of Implanted Fiducial Gold Markers is now standard procedure for external beam radiotherapy treatment of the prostate. Treatment margins can be reduced with image-based verification and correction for the prostate position. This ensures precision targeted radiation and reduces the dose to surrounding normal tissues (e.g. rectal wall, bladder).
Gold markers are clearly visible on CT and MR scans and can be used for CT-MR fusion for improved delineation of the prostatic gland.
Posted by Nasser at 1:11 PM